DescriptionPostdoctoral Position Available: Humanized Mouse Models of HIV Neuroinflammation and CNS HIV Persistence
A postdoctoral associate position is available to study HIV-associated neuroinflammation and CNS HIV persistence using advanced humanized mouse models. This position is supported by a newly funded NIH award focused on targeting the NLRP3 inflammasome as a driver of neuroinflammation bloodbrain barrier dysfunction and latent HIV reservoirs in the central nervous system.
The postdoctoral fellow will lead projects using state-of-the-art humanized mouse models of CNS HIV infection to define how innate immune signaling and inflammasome activation in microglia regulate neuroinflammation BBB integrity and HIV latency under antiretroviral therapy.
ResponsibilitiesResearch will include:
- Humanized mouse models of CNS HIV infection
- In vivo immunology and neuroimmune interactions
- HIV virology and latency in tissue reservoirs
- Inflammasome biology and innate immune signaling
- Viral and host immune phenotyping in brain and peripheral tissues
- Translational testing of immunomodulatory and antiviral strategies
The position is based at the Icahn School of Medicine at Mount Sinai in the Department of Medicine Division of Infectious Diseases and Immunology Institute.
QualificationsStrong candidates will have expertise in one or more of the following:
- HIV virology and latency
- Humanized mouse models
- In vivo immunology
- Neuroimmunology or CNS infection
- Viral pathogenesis and tissue reservoirs
A PhD and/or MD and a strong publication record are required.
Send CV and three references to:
Talia Swartz MD PhD Benjamin Chen MD PhD
Division of Infectious Diseases Division of Infectious Diseases
Department of Medicine Department of Medicine
Immunology Institute Immunology Institute
Icahn School of Medicine at Mount Sinai Icahn School of Medicine at Mount Sinai
@ @
DescriptionPostdoctoral Position Available: Humanized Mouse Models of HIV Neuroinflammation and CNS HIV PersistenceA postdoctoral associate position is available to study HIV-associated neuroinflammation and CNS HIV persistence using advanced humanized mouse models. This position is supported by a n...
DescriptionPostdoctoral Position Available: Humanized Mouse Models of HIV Neuroinflammation and CNS HIV Persistence
A postdoctoral associate position is available to study HIV-associated neuroinflammation and CNS HIV persistence using advanced humanized mouse models. This position is supported by a newly funded NIH award focused on targeting the NLRP3 inflammasome as a driver of neuroinflammation bloodbrain barrier dysfunction and latent HIV reservoirs in the central nervous system.
The postdoctoral fellow will lead projects using state-of-the-art humanized mouse models of CNS HIV infection to define how innate immune signaling and inflammasome activation in microglia regulate neuroinflammation BBB integrity and HIV latency under antiretroviral therapy.
ResponsibilitiesResearch will include:
- Humanized mouse models of CNS HIV infection
- In vivo immunology and neuroimmune interactions
- HIV virology and latency in tissue reservoirs
- Inflammasome biology and innate immune signaling
- Viral and host immune phenotyping in brain and peripheral tissues
- Translational testing of immunomodulatory and antiviral strategies
The position is based at the Icahn School of Medicine at Mount Sinai in the Department of Medicine Division of Infectious Diseases and Immunology Institute.
QualificationsStrong candidates will have expertise in one or more of the following:
- HIV virology and latency
- Humanized mouse models
- In vivo immunology
- Neuroimmunology or CNS infection
- Viral pathogenesis and tissue reservoirs
A PhD and/or MD and a strong publication record are required.
Send CV and three references to:
Talia Swartz MD PhD Benjamin Chen MD PhD
Division of Infectious Diseases Division of Infectious Diseases
Department of Medicine Department of Medicine
Immunology Institute Immunology Institute
Icahn School of Medicine at Mount Sinai Icahn School of Medicine at Mount Sinai
@ @
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